Objects related with this person

Potential mechanisms of resistance to cytarabine in AML patients.

In vivo mechanisms of resistance to cytarabine in acute myeloid leukaemia.

Nucleoside analogues and nucleobases in cancer treatment.

P-glycoprotein expression by cancer cells affects cell cytotoxicity and cell-cycle perturbations induced by six chemotherapeutic drugs.

Role of IMP-selective 5'-nucleotidase (cN-II) in hematological malignancies.

Pyrimidine nucleoside analogs in cancer treatment.

In vitro susceptibility of CD4(+) and CD8(+) T cell subsets to fludarabine.

Frameshift mutation in the Dok1 gene in chronic lymphocytic leukemia.

OSI-461 (OSI).

Characterization of a gemcitabine-resistant murine leukemic cell line: reversion of in vitro resistance by a mononucleotide prodrug.

Problems related to resistance to cytarabine in acute myeloid leukemia.

Quantitative analysis of nucleoside transporter and metabolism gene expression in chronic lymphocytic leukemia (CLL): identification of fludarabine-sensitive and -insensitive populations.

Histamine dihydrochloride (subcutaneous) Maxim.

[Metabolism, mechanism of action and resistance to cytotoxic nucleoside analogues]

Increased expression of the large subunit of ribonucleotide reductase is involved in resistance to gemcitabine in human mammary adenocarcinoma cells.

F-ara-AMP is a substrate of cytoplasmic 5'-nucleotidase II (cN-II): HPLC and NMR studies of enzymatic dephosphorylation.

Drug evaluation: sapacitabine--an orally available antimetabolite in the treatment of cancer.

Correlation of hENT1 expression and function with gemcitabine cytotoxicity in mantle cell lymphoma lines and clinical samples.



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